Current Fellows

Randi Parker

Randi Parker, MD (2015-2018)
Medical School: Ross University School of Medicine
Residency: Memorial University Medical Center, Savannah, GA
Research Interests: TBD

Deidra Ansah

Deidra Ansah, MD (2015-2018)
Medical School: Brody School of Medicine at East Carolina University
Residency: Emory University
Research Interests: TBD

Omar Rodriguez
Omar Rodriguez, MD (2014-2017)
Medical School: State University of New York Downstate Medical Center
Residency: North Shore LIJ - Staten Island
Chief Residency: North Shore LIJ - Staten Island
Research Interests: Thrombosis prevention in children with congenital heart disease: Thrombosis in children with systemic to pulmonary shunt is known and serious risk. I will be looking at bio markers and medication regiments in these children to determine if we can prevent thrombosis. I plan to expand this to other populations of children with congenital heart disease.

Derek Hoyme
Derek Hoyme, MD (2013-2016)
Medical School: University of Buffalo State University of New York School of Medicine and Biomedical Sciences
Residency: University of Arizona, Tucson, AZ
Research Interests: Resuscitation outcomes and outcomes of resuscitation education programs in Iowa High Schools.

Sonya Kirmani
Sonya Kirmani, MD (2013-2016)
Medical School: University of Iowa Roy J. and Lucille A. Carver College of Medicine
Residency: University of Iowa Hospitals and Clinics, Iowa City, IA
Research Interests: Dilated cardiomyopathy results in dilated ventricles with depressed systolic function. There are both idiopathic and familial forms, with most familial forms being autosomal dominant affecting structural genes of cardiomyocytes. It has been shown in mouse models that gene knockouts of Xin proteins, which co-localize with intercalated disk proteins N-cadherin and beta-catenin, result in a dilated cardiomyopathy phenotype. We have a large biobank of patients with dilated cardiomyopathy and are employing state-of-the-art sequencing techniques and analysis techniques to better understand how these genes may be implicated in the pathogenesis of dilated cardiomyopathy.